Matn1 cDNA ORF clone, Mus musculus(house mouse)

Interferon Regulatory Factor 6 Is Necessary for Salivary Glands and Pancreas Development.
Metwalli KA, Do MA, Nguyen K, Mallick S, Kin K, Farokhnia N, Jun G, Fakhouri WD
Journal of dental research97(2)226-236(2018 Feb)

Generation and characterization of mice with mesenchyme-specific deletion of the entire ESET histone methyltransferase protein.
Yang L, Gee AO, Zielinska-Kwiatkowska A, Chansky HA
Genesis (New York, N.Y. : 2000)(2017 Dec)

Cell origin, volume and arrangement are drivers of articular cartilage formation, morphogenesis and response to injury in mouse limbs.
Decker RS, Um HB, Dyment NA, Cottingham N, Usami Y, Enomoto-Iwamoto M, Kronenberg MS, Maye P, Rowe DW, Koyama E, Pacifici M
Developmental biology426(1)56-68(2017 06)

Absence of collagen IX accelerates hypertrophic differentiation in the embryonic mouse spine through a disturbance of the Ihh-PTHrP feedback loop.
Kamper M, Paulsson M, Zaucke F
Cell and tissue research367(2)359-367(2017 Feb)

Deficient Mechanical Activation of Anabolic Transcripts and Post-Traumatic Cartilage Degeneration in Matrilin-1 Knockout Mice.
Chen Y, Cossman J, Jayasuriya CT, Li X, Guan Y, Fonseca V, Yang K, Charbonneau C, Yu H, Kanbe K, Ma P, Darling E, Chen Q
PloS one11(6)e0156676(2016)

Results provide evidence that Matn1 plays a protective role in cartilage degeneration under altered mechanical environment.
Title: Deficient Mechanical Activation of Anabolic Transcripts and Post-Traumatic Cartilage Degeneration in Matrilin-1 Knockout Mice.

Matrilin-1 is an inhibitor of neovascularization
Title: Matrilin-1 is an inhibitor of neovascularization.

Secretion of matrilin 3 V194D mutant protein is not dependent on hetero-oligomerization with matrilin 1.
Title: Loss of matrilin 1 does not exacerbate the skeletal phenotype in a mouse model of multiple epiphyseal dysplasia caused by a Matn3 V194D mutation.

Transgenic mice expressing mutant (D469Delta) cartilage oligomeric matrix protein showed a slight gender specific growth retardation and both, tibial growth plate and sternum abnormalities.
Title: Transgenic mice expressing D469Delta mutated cartilage oligomeric matrix protein (COMP) show growth plate abnormalities and sternal malformations.

Data show that the cartilage matrix protein subdomain of type VII collagen is pathogenic for epidermolysis bullosa acquisita.
Title: The cartilage matrix protein subdomain of type VII collagen is pathogenic for epidermolysis bullosa acquisita.