Jun cDNA ORF clone, Rattus norvegicus(Norway rat)

NF??B regulates HSF1 and c?Jun activation in heat stress?induced intestinal epithelial cell apoptosis.
Li J, Liu Y, Duan P, Yu R, Gu Z, Li L, Liu Z, Su L
Molecular medicine reports17(2)3388-3396(2018 Feb)

A new miRNA regulator, miR-672, reduces cardiac hypertrophy by inhibiting JUN expression.
Lu Y, Wu F
Gene(2018 Jan)

Stimulation of B-Raf increases c-Jun and c-Fos expression and upregulates AP-1-regulated gene transcription in insulinoma cells.
Langfermann DS, R?ssler OG, Thiel G
Molecular and cellular endocrinology(2017 Dec)

These findings indicate that c-Jun induces PLCgamma1 expression transcriptionally and enhances rapid epithelial restitution after injury by activating Ca(2+) signal.
Title: c-Jun enhances intestinal epithelial restitution after wounding by increasing phospholipase C-?1 transcription.

In summary, our results suggest that HO-1 can decrease H/R-induced myocardiac cell apoptosis; the mechanism may be related to the activation of the Akt signaling pathway and, furthermore, to the inhibition of the JNK/c-Jun/caspase-3 signaling pathway.
Title: Heme oxygenase 1 induction protects myocardiac cells against hypoxia/reoxygenation-induced apoptosis : The role of JNK/c-Jun/Caspase-3 inhibition and Akt signaling enhancement.

The results suggest that an increase in the expression of PPARalpha/beta/gamma and RXRalpha as well as a decrease in AP-1 and importin-alpha3 expression/activity participates in the protective effect of 5,14-HEDGE against hypotension, tachycardia, and inflammation during endotoxemia and thus have a beneficial effect in septic shock treatment
Title: Contribution of PPAR?/?/?, AP-1, importin-?3, and RXR? to the protective effect of 5,14-HEDGE, a 20-HETE mimetic, against hypotension, tachycardia, and inflammation in a rat model of septic shock.

The aim of the present work was to assess the levels of mRNA encoded by genes c-jun and c-fos and the ratio of expression levels of these genes in various regions of the neonatal rat brain after the administration of dexamethasone
Title: [Influence of dexamethasone on the expression of immediate early genes c-fos and c-jun in different regions of the neonatal brain].

These results suggest that XT-1 expression is refractory to the disease process and to inhibition by inflammatory cytokines and that signaling through AP-1, Sp1, and Sp3 is important in the maintenance of XT-1 levels in NP cells.
Title: Xylosyltransferase-1 expression is refractory to inhibition by the inflammatory cytokines tumor necrosis factor ? and IL-1? in nucleus pulposus cells: novel regulation by AP-1, Sp1, and Sp3.