Calhm1 cDNA ORF clone, Mus musculus(house mouse)

Post-translational palmitoylation controls the voltage gating and lipid raft association of the CALHM1 channel.
Taruno A, Sun H, Nakajo K, Murakami T, Ohsaki Y, Kido MA, Ono F, Marunaka Y
The Journal of physiology595(18)6121-6145(2017 Sep)

CALHM1-Mediated ATP Release and Ciliary Beat Frequency Modulation in Nasal Epithelial Cells.
Workman AD, Carey RM, Chen B, Saunders CJ, Marambaud P, Mitchell CH, Tordoff MG, Lee RJ, Cohen NA
Scientific reports7(1)6687(2017 Jul)

Thus, CALHM1 controls synaptic activity in cerebral neurons and is required for the flexible processing of memory in mice.
Title: CALHM1 deficiency impairs cerebral neuron activity and memory flexibility in mice.

Abeta level reduction by CALHM1 could be explained by an increase in extracellular Abeta degradation by insulin-degrading enzyme.
Title: CALHM1 ion channel elicits amyloid-? clearance by insulin-degrading enzyme in cell lines and in vivo in the mouse brain.

Multiple transduction pathways make complex, concentration-dependent contributions to salty taste perception. One of these pathways depends on CALHM1 to detect hypertonic NaCl in the mouth and signal the aversive taste of concentrated salt.
Title: Salty taste deficits in CALHM1 knockout mice.

This study demonistrated that the Calhm1 knockot mice consumed only half as much sugar per day as did Wild type mice.
Title: Sucrose-conditioned flavor preferences in sweet ageusic T1r3 and Calhm1 knockout mice.

CALHM1 is a voltage-gated ATP-release channel required for sweet, bitter and umami taste perception
Title: CALHM1 ion channel mediates purinergic neurotransmission of sweet, bitter and umami tastes.