Btg2 cDNA ORF clone, Mus musculus(house mouse)

Epigenome profiling and editing of neocortical progenitor cells during development.
Albert M, Kalebic N, Florio M, Lakshmanaperumal N, Haffner C, Brandl H, Henry I, Huttner WB
The EMBO journal36(17)2642-2658(2017 09)

Systematic screening for skin, hair, and nail abnormalities in a large-scale knockout mouse program.
Sundberg JP, Dadras SS, Silva KA, Kennedy VE, Garland G, Murray SA, Sundberg BA, Schofield PN, Pratt CH
PloS one12(7)e0180682(2017)

Results identify a key molecular mechanism by which the BTG2-PRMT1 module regulates pre-B cell differentiation and inhibits pre-B cell leukemogenesis.
Title: The BTG2-PRMT1 module limits pre-B cell expansion by regulating the CDK4-Cyclin-D3 complex.

Results suggest that Btg2 is an anti-adipogenic factor and its expression is controlled by the Stat3 signaling pathway.
Title: B cell translocation gene 2 (Btg2) is regulated by Stat3 signaling and inhibits adipocyte differentiation.

Btg2 overexpression in vivo and in vitro induced all the observed changes during interdigit regression, including oxidative stress, arrest of cell cycle progression, transcriptional regulation of senescence markers, and caspase-mediated apoptosis.
Title: Apoptosis during embryonic tissue remodeling is accompanied by cell senescence.

study provides a novel molecular mechanism of BTG2-mediated induction of hepcidin gene expression, thereby contributing to a better understanding of the hepatic hepcidin production involved in iron homeostasis
Title: B-cell translocation gene 2 promotes hepatic hepcidin production via induction of Yin Yang 1.

these data show that both Btg1 and Btg2 are required for normal vertebral patterning of the axial skeleton, but each gene contributes differently in specifying the identity along the anterior-posterior axis of the skeleton.
Title: Targeted Deletion of Btg1 and Btg2 Results in Homeotic Transformation of the Axial Skeleton.